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Third, the data of Figure 4 show fold-changes compared to wild-type: even if strongly induced at the protein level, low abundance enzymes may not make a marked contribution to GSH oxidation within the cellular context

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The PI3K/Akt pathway also regulates the translocation of glucose transporter 4 (GLUT4) from intracellular vesicles to muscle and fat cell membranes, facilitating glucose uptake upon insulin stimulation

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